Sildenafil is often a potent and selective inhibitor of cGMP-distinct phosphodiesterase kind 5 (PDE5), and that is answerable for degradation of cGMP in the corpus cavernosum. The molecular framework of sildenafil is similar to that of cGMP and acts like a aggressive binding agent of PDE5 in the corpus cavernosum, resulting in far more cGMP and gre